“We experience moments absolutely free from worry. These brief respites are called panic.”
Don’t fight anxiety – use it. Equally, don’t fight resentment – use it as fuel.
Exercise is my way of coping with anxiety. I feel pretty terrible unless I do at least 30 minutes a day. Stretching, cardio, weights – it doesn’t really matter but you need to vent it – so do it positively.
If you don’t vent your feeling of stress & anxiety in a positive way, it is likely you will:
Project your stress onto someone else
Suffer from ‘internalising’ it – with IBS, stiff muscles, autoimmune issues
Both are shite. By the way, magnesium (including epsom salts for baths) is amazing for muscle stiffness and pain.
Anxiety Does Have Benefits
Chronic anxiety can make you feel very tired at certain points – your hypervigalance to danger can leave you exhausted at the end of the day. However, if you can think about something specific that makes you anxious, or someone that you don’t like very much – this can give you the energy to get up and do something – ideally exercise.
The best way to deal with anxiety, for me anyway, is to embrace and use it. I’ve literally shouted “oh f*ck off” to anxious thoughts in the past, but like a schoolyard bully; reacting like this just makes it more relentless. If you think of the ‘benefits’ and embrace it – thereby, not really reacting to it – it often fades.
*I imagine this is only useful advice for those with mild to moderate anxiety. So take it with a pinch of salt.
As much as I love a good meme or ‘Most people won’t share this’ post on social media; I’m not sure how helpful the whole mental health awareness thing is.
It definitely has some positives, but the real message should be around showing compassion and tolerance to everyone – not just people with a clinical diagnosis of depression and/or anxiety.
This goes for those who have mental health problems too – try and be nice. In my experience some people with depression are right horrible bastards (a bit like Ricky Gervais at the beginning of After Life).
I’m currently taking an SSRI anti-depressant called Sertraline. I came off my previous anti-depressant, because, although it was pretty effective at levelling my mood and stopping any suicidal thoughts etc. it proper fucked my memory.
A poor memory is a symptom of anxiety alone, and also some SSRI medication too. Put those two bad boys together and your memory is likely to be proper fucked.
If You Can’t Have Empathy – Have Compassion
Empathy requires some similar experience. If you’ve never had issues with mental health or your memory then it’s hard to put yourself in the other person’s shoes.
However, you don’t necessarily need empathy – just have some compassion.
Remember, people are just a product of their environment and genetics; nobody chooses to be stupid, dopey, or weird. I sure as fuck didn’t and wouldn’t be that way if I can help it!
What I really don’t like – is the look of bemusement some people give me when I can’t remember something. Taking the piss is fine, but I just want to chin people when they give me ‘the look of disdain’.
It’s a bit of a vicious circle of –
having a bad memory – and then hating yourself for forgetting stuff – compounded by other people losing it or mocking your for being forgetful.
Just try and be tolerant with people who are different, or specifically in my case, a bit eccentric + have memory problems. And definitely don’t get angry, especially in an environment like an office.
“Keep this thought handy when you feel a fit of rage coming on—it isn’t manly to be enraged. Rather, gentleness and civility are more human, and therefore manlier. A real man doesn’t give way to anger and discontent, and such a person has strength, courage, and endurance—unlike the angry and complaining. The nearer a man comes to a calm mind, the closer he is to strength.” – Marcus Aurelius
How to Not be a C*nt
Feedback in private & praise in public. Don’t be one of those passive aggressive office-wankers who likes to feedback mistakes in front of everyone
Don’t slag people off unless they’ve done something horrendous
Accept people as they are
Don’t make fun of people in front of an audience or people you don’t know very well – that’s when banter goes too far IMO
Have patience with people – especially those who have issues with their memories as this can be a symptom of anxiety and/or SSRIs
By the way – I think it’s fine to tell a depressed person to stop complaining. Depression is as contagious as man flu; if someone is constantly complaining and blaming other people for all their problems – please – tell them to shut up. Unfortunately this often leads to the victim-triad; by being told not to be a victim, people often feel like you’re being unfair; making them even more of a victim. So good luck with that issue.
Stuff that Helps if You Do Have Mental Health Problems
Laugh At Yourself
If you do have mental health problems – I’ve found ‘thoughts of the self’ are often the big issue. Remember not to take life so seriously.
We’re all going to die and nobody will remember us – so fuck it
Thinking of and looking out for other people is actually the best way to stop thinking about yourself. Also, remember that 99.9% people aren’t thinking about you (they’re probably thinking about themselves), so don’t stress about it!
Brainwash Yourself with Positive Music, Videos & People
I listen to a video playlist in the morning, that includes the likes of Jocko Willink, David Goggins and positive affirmations. It’s hard to feel negative after I’ve listened to it. Also – avoid the news! Turn that shite off.
The Logical Way to Think – Isn’t Always the Best Way to Think
Be cynical but don’t suck the joy out of everything with it. Sometimes, it’s best to choose another chain of thought.
For example, it might be helpful to take ownership/accountability of all your problems, even if nothing was really your fault. Fuck playing the victim – that shit never helps.
For example, let’s say you pay a tradesman up front and he doesn’t show up – is that your fault or the tradesman’s?
I don’t think that example explains the concept very well, so if you have 10 mins, watch the video below:
Another, probably better example – if you watch Lord of the Rings thinking “this is made up stupid shite”, then you might be right, but it’s still not a ‘good’ way to think – especially if you’re effecting people around you, who may be enjoying it.
I guess religion and faith is another example – it might not be logical, but if it gives you or other people strength and courage, then let them get on with it.
Remember too, you tend to get back what you ‘put out’ into the world. You can also develop a habit of positive (and negative thinking). Make an effort to be positive everyday and it does get easier!
Nothing to do with MMA – sorry – however, I thought this would be very useful for educating the kids whilst going on one of our very common walks in the woods and countryside. It’s also interesting for me to find out and have a future reference.
Most Common UK Trees
Aspen – Populus Tremula
The leaves are easily damaged by sunlight.
The top leaves move a lot, so the lower leaves get their fair share of sunlight
The trees can be interconnected underground – the same tree is effectively produces several trees all connected together.
Silver Birch – Betula Pendula
Birch can live to about 80 years old.
The bark sheds layers, like skin
Leaves are small and triangular with a toothed edge.
Prefer light, dry and acidic soils.
Sessile Oak – Quercus Petraea
Unlike the English Oak, the sessile oak does not have stalks on its acorns.
These trees can be identified by their lobed shaped leaves and their acorns.
The greyish bark of this oak, used to be used in leather tanning industries.
Sweet Chestnut – Castanea Sativa
Sweet chestnut are completely different to horse chestnut, so don’t get them confused.
The easiest way to identify a sweet chestnut is by it’s big, bold, spear-shaped leaves.
The leaves have a serrated edge, like Rambo’s knife
A great demonstration of strength of the hands, grip and the ability to brace against your leg – bending horseshoes is a great party trick (if you party in a barn) and also a good goal to aim for if you are a strongman or someone who is looking to increase grip strength.
Know Your Horseshoes Before You Attempt to Bend it
A horseshoe is only as strong as it’s weakest point – a bit like a chain.
Some horseshoes have ‘fullers’. Fullers are the grooves that allow for the insertion of nails. Some horseshoes have deeper & wider fullers than others – from a strongman perspective, this can weaken the horseshoe significantly – making it easier to bend.
Technical Tips for Horseshoe Bending
The Strength comes from the triceps at the end of their range-of-motion. Known as “lockout strength”, the more extended and near full extension your arms are, the stronger force you will generate against the horseshoe.
Padding or Not to Pad?
Padding the horseshoe may help with limiting damage and pressure on your hands but it will also dissipate some of the pressure on the horseshoe – certainly it will spread the pressure that would otherwise be isolated to a specific section of the horseshoe.
An abrasive material such as a (very) thick and wide bandaid/sticking plaster, or even a thick tissue, appears to be the best way to bend horseshoes without cutting your hands.
Twist and Shout
Referred to some as “the crushdown” – the rotational force of bodyweight, bracing from the shoulders and pushing from the triceps and arms is a universal movement in pretty much everyone on youtube seen bending a horseshoe.
A Word of Caution about HorseShoe Bending
This movement will put a lot of stress on your shoulders, elbows and wrists. The movement is partially (kind of) isometric – i.e. static, in that the horshoe moves very little or doesn’t move at all.
When a movement is isometric, the forces will ‘push back’ more on the body, causing more stress on the joints and ligaments.
You can see below, how by bracing and pushing down with his bodyweight and lowering his head, the abdominals come into play quite significantly:
Not everyone appears to incorporate the abs when bending however. This guy has more of a lateral movement:
Training to Bend Horseshoes
Firstly, if you want to bend horseshoes – you will need a horseshoe!
You will need a horse shoe to practice on, to build the specific type of strength required to bend it. Unless you use something very similar to bend, like a bar or rod etc and build up to a horseshoe
The SAID principle dictates it – Specific Adaptation to Imposed Demands.
Purchase one of the weaker horseshoes – with a fuller groove in it.
Here’s a website that sells horseshoes – ones that a great for starting off and achieving your first horseshoe bend!
St. Croix Ultra Lite are good for beginners. But still very difficult!
St Croix Polo are amongst the ‘second easiest’ level of horseshoe to bed.
Following on, go for a St. Croix Forge Lite Rim 10.
Training Grip Strength to Bend Horseshoes
A great way to build grip strength is to, well, grip things.
Chin ups are a good start for beginners, even just hanging from a bar as long as possible is a very effective way to build your general hand strength.
Bottom up kettlebell presses are also great for building grip strength – as you have to balance the weight as well as push it upwards.
Training with a sledgehammer, or even a broom can be effective too. Doing curls and extensions with the bar:
A word of caution about grip training – you should work you wrist extensors (muscle for gripping) as well as your wrist extensors – otherwise you are likely to get some nasty over-use and imbalance injuries such as golfer’s elbow.
Holding 1 or 2 weights plates for as long as possible is another great way to train grip.
Training Lockout Strength
Probably the best way to build lockout strength for horseshoe bending, is heavy dips on parallel bars.
Heavy bench press, using bench press blocks and/or bands or chains that make the resistance greater at the top of the movement, are also great tools & exercises for building lockout strength.
Abs are used in some way, pretty much regardless of your horseshoe bending technique. Hanging leg raises and weighted crunches will help develop specific core strength.
Using cables can make the exercise more specific to horseshoe bending. Kneel down and hold the cables overhead, as you crunch downward.
Boxing 🥊 is a very demanding sport. There are lots of aspects of fitness that go into conditioning for boxing – one crucial thing I want everyone to know however – is that chest-strength, does not equate to punching power!
Very little power if generated specifically from the chest. The whole body (or kinetic chain) needs to be worked.
In fact, the difference between ‘low level’ & ‘high level’ boxers in one study – was the generation of force from the legs when punching:
Training the chest in isolation from the rest of the body, will hinder, not help your boxing.
Some tips for training your chest whilst not interfering with boxing fitness include
Replicate punches with a small amount of resistance so that it doesn’t interfere with technique and proper punching mechanics. e.g. punches with cables or bands with a low amount of resistance
Train muscles for power & speed, not strength
Heavy bench press has been shown in studies to reduce shoulder mobility – this is bad for boxing
Do lots of shoulder and chest mobility work before & after doing weights
Train movements not muscles!
Modify some (not all) squat exercises so that you are in a boxing stance, rather than a standard square-on stance
Example Boxing Chest Workout
Dumbbell Bench Press
Plyo Press Ups – Side to Side
Medicine Ball Throw into Press Up
Thrusters – (Squat into Press)
Pads or punch bag
Warm up with 2 rounds of shadow boxing, 5 minutes of skipping and some shoulder mobility exercises.
Dumbbell Bench Press 2 sets of 8 Reps
Plyometric Press Ups ‘side to side’ medicine ball or BOSU – 2 sets of 10 Reps
Medicine Ball Throw into Press Up – 2 sets of 8 Reps
Band Punches 2 sets of 10 reps on each arm (total of 4 sets)
Thrusters – Squat & Press – 2 sets of 10 reps
Thrusters are a good ‘finisher’ that incorporate the entire body. These types of movements are important to include so that the chest doesn’t get ‘used to’ working in isolation from the legs and core.
Remember to work your back muscles equally with pulling exercises like one-arm-row, otherwise you will get a shoulder imbalance.
You can use TENs machines to prevent muscle loss whilst injured
Nutrition is important – Under-eating will lead to more muscle loss
Clenbuteral is an illegal performance enhancing drug that prevents muscle loss
Alpha Lipoic Acid supplementation may help prevent muscle loss and help to alleviate some of the effects of the loss of insulin sensitivity
No significant de-training effects occur if you can get back to training within 2 weeks
For information purposes only. Exercise at your own risk
Professional sport in the twenty first century has evolved into a competitive business. Immense financial investments into sports clubs, sports governing bodies and development centres, has seen athletes converted into commodities that are expected to deliver success. As sports clubs float on the stock market, athletes follow rigorous training regimes that push them to their physical and mental limit. MMA shares the pressures and expectations of any other top-level sport. As it verges on the brink of mainstream acceptance, it survives primarily as a form of entertainment, rather than participatory sport.
Pay per view and DVD sales underpin its survival, and as a result the combatants are expected to display feats of awesome athletic ability and technique, each time they enter the cage. Fuelled by massive economic investment, MMA training has developed into a scientific discipline that addresses and attends to every aspect of physical and mental fitness.
However, as boundaries in performance are continually broken and re-established, athletes increase their risk of ‘burnout’ or physical injury. Although the recruitment of sports psychologists and physiotherapists may reduce the likelihood of such an event, it is doubtful that a competitor will end his/her career without having to endure a serious injury or slump in performance at some point. Successful fighters find a balance between hard training and rest.
Rest is required so that adaptation to training may occur. Progression and consistency is key in any training schedule and injuries can set back athletes by months, even years. It is therefore crucial that athletes limit their risk of injury by avoiding over-training, and by acquiring knowledge concerning how best to limit the ‘detraining’ effect, so that an injury or rest period is managed correctly. The following report will examine the consequences of rest and injury upon different aspects of athletic performance, and will also discuss the best ways of inhibiting the detraining effect. A number of reports and research documents shall be examined and critiqued, so that by the end of the report the question ‘detraining: threat or therapy’ will be successfully discussed and answered.
Injury & Deconditioning
According to Quinn (2004), one of the five principles of conditioning is the ‘Principle of Use/Disuse’. Simplified, this would imply that your muscles hypertrophy with use and atrophy with disuse. However, this ‘rule’ should not be taken completely literally, as it is important to find a balance between training and rest. There must be periods of low intensity between periods of high intensity to allow for recovery and training adaptation to occur.
Thanks to recent research, deconditioning theories have become more clear and specific. Mujika et al (2000) studied well-conditioned athletes who had trained for year and then stopped exercise all together. After a three month rest from cycling training, researchers found that the athletes lost 57% of their aerobic conditioning.
However, Rietjens et al (2001) investigated the effects of reduced training on physical condition and performance in well-trained cyclists and whether an intermittent exercise programme would maintain physiological training adaptations.
Neither group showed changes in maximal workload for the Continuous Training and Intermittent Training group respectively, and it was concluded that well trained cyclists who reduce training intensity and volume for 21 days can maintain physiological adaptations, as measured during submaximal and maximal exercise. In addition the study seem to suggest that an intermittent training regimen has no advantage over a continuous training regimen during a detraining period. It would appear that a certain level of exercise is required to maintain sport specific fitness.
However, the intensity and duration of this ‘level of activity’ remains controversial and is specific to each sport. The level of activity necessary will depend upon an athlete’s baseline fitness level and sport (Winters & Snow, 2000). However, it would appear that if an athlete can maintain some exercise on a weekly basis, he/she could retain a greater percentage of his/her baseline fitness levels. Although it is not always possible, athletes should attempt to adapt their training in accordance to an injury.
Cross training through an injury is always an option, for example ‘aqua-jogging’ is an effective activity that enables runners to maintain fitness levels whilst recovering from repetitive strain injuries to the knees, shins and/or back (http://sportsmedicine.about.com/cs/exercisephysiology/a/aa073003a.htm). Repetitive strain injuries are increasingly common among top athletes (Smith, 2003). Many top athletes train 3-5 hours per day in an attempt to improve their level of conditioning and skill.
Physical activity is a way of life for these people, and can dominate their thoughts and actions for a number of years. When an individual stops participating in high levels of physical activity, the physiological changes that occur are usual referred to as ‘detraining’. This often occurs when an athlete becomes injured. Swain et al (1994) and Cooper (1982) suggest that a few days of rest or inactivity may enhance performance and physiological adaptations to training. However there is a critical, individual specific, period of rest, where performance capability begins to decrease. It would also appear that different aspects of fitness deteriorate at different rates, and all levels of detraining depend of training history. For example, within a few days, a person’s insulin sensitivity is decreased, whereas muscular strength can take weeks to deteriorate (Taafe & Marcus, 1997).
Skeletal Muscle Size, Strength & Power
Sports such as powerlifting and rugby predominantly involve short, explosive movements. However most sports, such as wrestling require high levels of endurance and several other specific types of fitness and skill. Strength training may therefore, not always remain as a top priority, and may have to be maintained whilst other aspects of performance are targeted for improvement. Haggmark et al (1986) suggest that continuing to train once every 10 to 14 days, athletes can maintain strength and power for up to 4 months.
This may be important for the injured athlete, who cannot train at maximal intensity. The real losses in muscle mass and strength occur, not when training per se is stopped, but when a joint is completely immobilized. Daily activities are enough to reduce any sudden loss in performance capacity, and inhibit the drastic losses that occur through complete immobilisation.
Skeletal muscle undergoes a substantial decrease in size once a certain limb or area of the body becomes inactive. This is known as atrophy, and is accompanied by a considerable loss in power and strength. Total inactivity results in rapid losses, whilst prolonged periods of reduced activity can result in more gradual losses that become quite significant.
Taaffe and Marcus (1997) investigated the effects of cessation (8 weeks) and subsequent resumption (12 weeks) of training on muscle strength in elderly men, after completing 24 weeks of resistance training. The results indicate that elderly men lose some muscle strength following short-term detraining, but that only a brief period of retraining suffices to regain maximal strength. Reduction of fibre cross-sectional area with detraining, suggests that much of the retention in strength with detraining and reacquisition of lost strength with retraining reflects neural adaptation.
The strength that was maintained could be accounted for by neuro-muscular adaptation that occurs in the first weeks of adoption of a weight training programme; this relates to the acquisition of technical correctness and the ability to recruit the maximal number of muscle fibres with correct timing. One of the very first studies to investigate detraining (McMorris et al, 1954) showed that 45% of strength gains from a 12 weeks resistance training programme, were maintained following a 1 year absence from training. It has been suggested that this may also reflect retention of neuromuscular functioning (Housh et al, 1995). A study conducted by Costill et al (1998) upon university swimmers showed that a 4 week rest from activity did not affect shoulder or arm strength, however, swimming power was reduced by approximately 10% whether the swimmers underwent complete rest or trained at a reduced frequency of one session per week.
This suggests that functional strength and/or power may be reduced at an accelerated rate compared with strength per se. This study points to a possible area of inaccuracy in other studies – measurement techniques. The strength was measured ‘on land’ with a biokinetic swim bench, whilst power was measured in the water, using tethered swimming that allows the swimmers to use natural actions. It would appear therefore that the less specific land measurements may not reflect any effect upon performance.
When muscles are not used in the specific way that accurately replicates performance, neurological control appears to be reduced (GSSI, 2001 Conference) and normal fibre recruitment is hindered. Rest is an essential component of strength training which allows for muscular adaptation to a training load. Strength can be maintained a lot easier than it is acquired (Haggmark et al, 1986). Coaches and trainers should take this into account when designing periodised macro cycles which are tailored to the specific needs of a sport. Fitness tests such as the standing jump test and varies one repetition max tests should be used at intervals during the season in order to monitor fitness levels to ensure that each aspect of conditioning is either being maintained or improved.
Muscular endurance decreases after just 2 weeks of inactivity (Winters & Snow, 2000). Skeletal muscle is characterized by its ability to dynamically adapt to variable levels of functional demands. During periods of insufficient training stimulus, muscular endurance decreases rapidly. Initial deterioration of endurance may be due to a decreased capillary density, which could take place within 2–3 wk of inactivity. Arterial-venous oxygen difference declines if training stoppage continues beyond 3–8 wk. Rapid and progressive reductions in oxidative enzyme activities bring about a reduced mitochondrial ATP production (Winters & Snow, 2000). The above changes are related to the reduction in VO2max observed during long-term training cessation.
These muscular characteristics remain above sedentary values in the detrained athlete but usually return to baseline values in recently trained individuals. Glycolytic enzyme activities show non-systematic changes during periods of training cessation. Fibre distribution remains unchanged during the initial weeks of inactivity, but oxidative fibres may decrease in endurance athletes and increase in strength-trained athletes within 8 wk of training stoppage. At this time, not enough evidence is available to determine, whether this performance decrement results from changes in the muscle or from changes in cardiovascular capacity (Coyle, 1984). Studies have shown that after a week of cast immobilisation, the activities of oxidative enzymes such as succinate dehydrogenase and cytochrome oxidase decrease by 40 to 60%. In contrast, when athletes stop training, the activities of the muscles’ glycolytic enzymes, such as phosphofructokinase, change little, if at all, for at least 4 weeks.
Coyle et al (1984) observed no change in glycolytic enzyme activity with up to 84 days of inactivity, but an average fall of over 50% in the activity of oxidative enzymes. Despite the apparent resilience of glycolytic enzymes to detrimental effects of detraining, muscle glycogen content decreases. This may be due to a sharp decrease in muscle glycogen content. Costill et al (1985) observed a 40% reduction in muscle glycogen content following 4 weeks ‘detraining’. These values were virtually identical to a totally untrained individual. This may be due to a decrease in insulin sensitivity and GLUT 4 receptor activity (Costill, 1985).
V02Max and Endurance Performance
Following complete cessation of training, VO2max decreases in previously highly trained individuals after as little as 4 weeks, with the decrease in VO2max varying between 4 and 14% (Coyle, 1984). The decrease in VO2max during the first 3 weeks of detraining is due to a decrease in maximal cardiac output. Subsequent decreases in VO2max are due to decreased oxygen extraction, likely from a decrease in mitochondrial density (Houmard et al, 1989) Petibois and Déléris (2003), investigated changes in the metabolic response to an endurance exercise (18 rowing km at 75 % of maximal aerobic velocity) during detraining in ten rowers previously highly-trained. Maximal aerobic velocity (VO2 max) and the metabolic response to exercise were determined in the 1st, 24th, and 47th week (training), and in the 52nd, 76th, and 99th week (detraining). Short-term detraining (5 weeks) resulted in a lower adipose tissue triglyceride (TG) delivery during exercise (p = 0.029), but this one did not represent a direct metabolic limit to exercise since the liver TG delivery increased (p = 0.039), total fatty acid concentration remained unchanged. Long-term detraining (52 weeks) altered even more the metabolic response to exercise with a decreased total fatty acid concentration during exercise (week 99: 10.6 +/- 2.0 mmol/l; p = 0.022), which induced a higher glycolysis utilization.
A haemolytic response to endurance exercise was observed through haptoglobin and transferrin concentration changes (weeks 47 vs. 99; p = 0.029 and 0.027, respectively), which resulted probably from higher red blood cell destruction. Endurance-trained athletes should avoid detraining periods over a few weeks since alterations of the metabolic adaptations to training may become rapidly chronic after such delays. (Petibois C & Deleris G, 2003).
Saltin et al (1968), examined the effect of 20 days of bed rest upon cardio respiratory endurance. On average the subjects experienced a 25% decrease in submaximal stroke volume, a 25% decrease in maximal cardiac output, and 27% decrease in maximal oxygen consumption. The reductions in cardiac output and V02Max appear to be related to the decrease in stroke volume, which in turn is probably due to a decrease in heart volume, total blood volume and ventricular contractibility.
The trained subjects experienced decreases in endurance compared to the untrained subjects; furthermore the untrained subjects regained their baseline fitness in 10 days, compared to 40 days for the trained subjects. This drop in conditioning may have implications for the ‘off season’ in many sports. Athletes should either maintain physical activity during the breaks from competition, or the pre season should begin over 40 days prior to the beginning of the competitive season. Giada et al (1998) studied 12 young and 12 older healthy sedentary males to examine the effects of training, and detraining on men of different age groups. Each subject underwent a maximal exercise test using a cycle-ergometer in order to measure maximum oxygen consumption and a echocardiography in order to assess left ventricle morphology and systolic function.
During the training period both groups of athletes showed higher values of maximum oxygen consumption, left ventricular wall thickness, end-diastolic diameter and volume, as well as left ventricular mass, than their control subjects. After the detraining period the wall thickness decreased only in young athletes, while left ventricular mass and end-diastolic diameter and volume reduced only in older athletes. It would appear from this study that the detraining effect may have many variables, one of which is age. However, further research using a greater number of subjects and control groups is warranted to provide a conclusive answer. Impairment of cardiovascular function following a few weeks of detraining is largely caused b a reduction in plasma volume, which in turn diminishes the plasma volume of the heart. Coyle et al (1986) observed a 9% decrease in blood volume and a 12% decrease in both stroke volume and plasma volume after two to four weeks of detraining following sustained periods of training, either running or cycling.
After the subjects were detrained, they were infused with dextrin solution to expand their blood volume until it exceeded their trained level. This improved cardiovascular function and V02max, but had little positive effect upon endurance performance. Reductions in cardio respiratory endurance appear to be much greater than reductions in strength and power over identical periods of detraining. Drinkwater and Horvarth (1972) studied seven female track athletes and again 3 months after training had ended. During the 3 month period, the athletes participated in typical physical activities for their age group, including physical education. At the end of the 3 months their V02Max had decreased by an average of 15.5%. Their new V02Max levels were similar to that of non-athletic girls the same age. Despite the rapid losses outlined in these studies, it would appear that a detraining effect only occurs if a person decreases their training volume by one thirds or more (Houmard et al, 1989), and/or their intensity falls below 70% Vo2Max.
Training for endurance or endurance sports differs from power and strength training in that it can be, and often is trained almost everyday (www.pponline.co.uk). Endurance can decrease in a matter of days after training has stopped. For example it has been shown that insulin sensitivity decreases to sedentary levels after just 10 days of rest (Gautier, 2004), leading to lower levels of muscle and liver glycogen. Athletes should include a period of reduced training intensity and frequency, but it is recommended that rest does not exceed 10 days and intensity does not fall below 70% V02Max.
Other Physiological Changes
Herd et al (1998) examined the effect of detraining upon postprandial lipeamia. Fourteen normolipidaemic, recreationally active young adults aged 18-31 years participated, in two self-selected groups: three men and five women (BMI 21·7-27·6 kg/m2) completed 13 weeks of running training, after which they refrained from exercise for 9 d; three men and three women (BMI 21·5-25·6 kg/m2) maintained their usual lifestyle. Oral fat tolerance tests were conducted at baseline and again 15 h, 60 h and 9 d after the runners’ last training session. In the absence of the acute effect of exercise, i.e. 60 h after the last training session, there was no effect of training on either postprandial lipaemia or on post-heparin lipo protein lipase activity. However, changes during 9 d of detraining in both these variables differed significantly between groups; after 2 d without exercise (60 h test), the runners’ lipaemic response was 37% higher than it was the morning after their last training session (15 h test; runners v. controls P , 0·05), with a reciprocal decrease in post-heparin LPL activity (P < 0:01). These findings suggest that improved fitness does not necessarily confer an effect on postprandial lipaemia above that attributable to a single session of exercise. Postprandial lipaemia is not elevated with training status and is therefore not influenced by detraining. It is however an acute response, and training must be regular and consistent in order to experience any benefits.
Retraining After Injury
Recovery of conditioning after a period of inactivity is affected by a person’s fitness level, length of training before the period of inactivity, and length of inactivity. Those who were fittest before inactivity suffer the greatest losses in fitness and also take longest to recover from inactivity. Two or three weeks of detraining have shown to cause the following decrements in highly trained subjects (Ross & Leveritt, 2001): Muscle oxidative enzymes decreased by 13 to 24% Performance time decreased by about 2 to 5% Vo2 max decreased by about 4% In a study conducted by Saltin et al (1968), 3 weeks of detraining resulted in significant losses in performance capacity. Following 15 days of retraining, only Vo2max had returned to its original trained level. Oxidative enzymes did not improve and although performance time showed some improvement, it still remained 2 to 5% below the trained time. This suggests that in elite athletes, the duration of retraining must exceed that of detraining in order to re-establish performance capacity to its original values.
Nutrition is an important consideration during times of inactivity. For example, a rugby player should decide whether or not to remain in positive energy balance in order to prevent further catabolism of muscle tissue, or should he try and remain as close as possible to a neutral energy balance in order to offset accumulation of fat tissue.
Van Baak (2004) showed that underfeeding by 250kcal per day over 6 weeks, resulted in the catabolism of 75% body fat and 25% fat free mass; underfeeding in addition to inactivity may result in massive in strength and power for the rugby player. Alpha Lipoic Acid mimics (to a certain extent) the anabolic effects of insulin (Eason et al, 2002). Loss of insulin resistance is on of the major causes of deconditioning during detraining, and therefore this supplement may be useful to injured athletes by preventing the unwanted accumulation of body fat, whilst keeping muscle lipid and glycogen levels high.
PEDs & Injury Recovery
Many athletes resort to illegal substance use in an attempt to offset the effects of inactivity or to accelerate the recovery process. Clenbuterol, a bronchodilator that used to be prescribed to asthmatics, is believed to poses anticatabolic properties, and is often used in the post cycle stages by steroid taking athletes, in order to avoid the catabolic effects of inhibited testosterone levels. Clinical research conducted by Montovani et al, 2001 has supported the theory that clenbuterol does possess some anti catabolic properties.
Anabolic steroids themselves have also been used by injured athletes in the past. Anabolic-androgenic steroids increase protein synthesis and therefore may mimic the effects of training, and repair tissue damage at an accelerated rate. Primabolin and stanozol are reputed as possessing the greatest anti catabolic properties, but stanozol, as a non-aromitising steroid (does not convert to oestrogen), is said to have a possible damaging effect upon joints by decreasing their fluid content. Conversely, aromatising steroids such as deca durabolin or deca nandrolone, are said to accelerate recovery from joint injuries by increasing the levels of synovial fluid within the joint capsule (MIMS, 2003).
TENS / NMES Electrical Stimulation for Injuries
Electrical Stimulation In one of the earliest published studies on the effects of high-intensity neuro-muscular electrical stimulation (NMES) on the maintenance of size and strength in immobilised muscles, researchers electrically stimulated the quadriceps and hamstrings on a daily basis for three weeks in the immobilised leg of an athlete wearing a lower-extremity cast as a result of Grade-II medial-collateral and anterior-cruciate ligament sprains in his knee (Pocari et al, 2003; Caggiano et al, 1994). On the day the cast was removed, the girth of the athlete’s thigh was increased, suggesting that muscle hypertrophy had occurred, instead of the usual cast-associated atrophy. In addition, single-leg, vertical-leap height was 92% as great in the immobilised leg following cast removal, compared with the uninjured leg, and the athlete was able to immediately return to competition.
The use of electrical stimulation to prevent muscle atrophy as a result of prolonged knee immobilisation following either injury or knee-ligament reconstructive surgery has been very intensely studied. Research (e.g. Pocari et al, 2003) has shown that electrical stimulation is effective in preventing decreases in muscle strength, muscle size, and even the oxygen-consumption capabilities of thigh muscles after knee immobilisation. In all but one of the studies in this area which have been published in scientific journals, electrical stimulation has been shown to be better in preventing negative changes in leg and knee-joint function, compared with no exercise, isometric exercise of the quadriceps-femoris muscles, and even isometric co-contractions of the quadriceps and hamstrings.
Studies suggested that these physiological and metabolic changes started after the 4-7th days of NMES application (Hudlicka et al, 1984) Type 2 fibrils prominently change to type 1 fibrils which are more rich for mitochondrial content, capillary density and oxidative enzyme capacity after electrical stimulation application, while the hypertrophy and hyperplasia in type 2 fibrils are more prominent after Isometric Exercise sessions (Cabric et al, 1998). This would suggest that isometric exercise may be of greater value to power athletes during injury, and electrical stimulation may be more important to endurance athletes.
Conclusion This report has illustrated the way in which different aspects of fitness tend to deteriorate with inactivity or reduced training. It can be inferred from this that performance level in different sports will diminish at a rate determine by its specific demands.
However, over-training remains a problem with elite athletes, and it is generally agreed that an of-season period is required within which training intensity should be significantly reduced in order to maintain a high level of fitness without excessive strain upon the body (Smith, 2003, Roden, 2004).
Over-training can result in high levels of cortisol, free radicals and musculo-skeletal injuries. This will lead to a slow recovery from training sessions, susceptibility to illnesses and disease and ultimately, a decreased performance. Over-training symptoms include decreased appetite and body weight loss, muscle tenderness, head colds, nausea, sleep disturbances and elevated blood pressure (Willmore & Costill, 1999). This report concludes that injured athletes should attempt to train around injuries where possible, and take into consideration the possible influence that nutrition may have over their recovery.
Metabolic Responses to Exercise in the Heat – Scientific Analysis
For information purposes only. Exercise at your own risk
Any team or athlete that competes on an international stage will no doubt have to deal with the physiological challenges that extreme environments can present them with. Competing in extreme heat, is beyond a challenge, it can be life threatening.
Heat is transferred from the core, via the blood to the skin, and then to the environment via:
Conduction: heat transfer from one material to another through direct molecular contact e.g. skin surface to clothing
Convection: Transferring heat by the motion of a gas or liquid e.g. skin to moving air
Radiation: heat transfer in the ford of infrared rays, e.g. heat gain by sun exposure
Evaporation: as fluid evaporates, usually sweat, heat is lost (approx 80% of total heat loss when active).
Heat exchange is controlled in the brain.
Thermoreceptors in the hypothalamus monitor blood temperature as it flows through the brain. Peripheral receptors in the skin monitor environmental temperature providing information to the hypothalamus and cerebral cortex allowing behavioural changes to be made.
For example, hypothalamus signals sweat glands to secret sweat when it has determeined that the blood temperature is high. Smooth muscle around arterioles ma also become dilated, increasing blood flow to the skin, and in turn, heat lost to the environment from the body.
Exercise in the heat results in a reduction in endurance and in intermittent exercise performance. According to Maughan (1992), loses as small as 2% of boy mass impairs performance. A study conducted by Suzuki et al, showed that cycling time to exhaustion at 66% of max aerobic capacity, was 91 minutes at 0 degress Celsius and 19 minutes at 40 degrees Celsius, whilst Adams et al showed that a well train runner was exhausted after 165 minutes at 10 celsius, compared to 102 minutes at 35 celsius.
There are a number of ‘problems’ that the body faces when exercising in the heat. There is competition between active muscles and skin for a limited blood supply. Heart rate increases to compensate for the decreased stroke volume. Redistribution of blood reduces the amount of blood that returns to the heart. Inability of the body to meet all the demands for blood flow results in hyperthermia. In addition, reduced blood supply to the muscle may result in greater reliance on muscle glycogen and anaerobic energy production, resulting in a greater and more immediate accumulation of blood lactate.
In addition to this, an individual’s body temperature may also be influenced by hormone fluctuations. For example, there can be a sharp elevation in core temperature (0.5-0.75 celsius) on day 14 of a menstrual cycle.
Stay Hydrated Whilst Exercising in the Heat
For every litre of sweat lost, rectal temperature increases by 0.3 C, cardiac output decreases by 1Litre/min, heart rate increases by 8 beats/minu and perceived rate of exertion increases rapidly (Coyle and Montain (1992)). The best way to attempt to stay rehydrated is to drink small amounts frequently. Drinking frequently keeps gastric emptying more rapid, which helps increase rehydration. Drinking about 5ml/kg initially, followed by 2ml/kg every 15 to 20 minutes of exercise helps to keep gastric emptying high.
In addition to drinking small and frequent amounts of fluid, adding sodium and glucose can enhance rehydration. Water crosses the small intestine passively, whereas, sodium and glucose move across via active transport, and ‘pull’ water with them. Consuming a 5% glucose drink, has also been clinically proven to help reduce the reduction in a person’s immune function, as seen after intense exercise.
Dehydration and heat exhaustion can certainly be limiting factors in human performance. In addition, severe cases of either can be fatal. Drink small amounts of a 5% glucose drink often. It is also wise to weigh oneself before an event or training session, and then again after. Replace 150% of the weight lost; so if you lose 2kg, drink 1.5L of water. Again, a glucose solution is preferable, as this will also help to replace muscle glycogen.
This article will look at the most common knee injuries that can arise from both MMA training and competition.
Anatomy of the Knee
The knee joint works like a hinge to bend and straighten the lower leg and is made up of the femur (thigh bone), the tibia (shin bone) and the patella (knee cap). Between the tibia and femur is the meniscus that acts as a shock absorber distributing weight and improves joint stability. The meniscus is what most people consider to be the “cartilage” of the knee. The majority of strength and stability of the knee is provided by ligaments which are strong, elastic bands of tissue that connect bone to bone.
Knee Injuries Due to its structure and its role in weight bearing the knee is a common site for injury in contact sports and activities that rely on rapid rotation during weight bearing such as MMA.
As with all other joints, knee injuries may be divided into 3 categories: · Single-event (acute) trauma · Acute-on-chronic injury · Multiple-repetition (or chronic) injuries.
The principles for management of each type of injury have been covered in a previous article so here we’ll concentrate of specific injuries. The most common injuries for a MMA fighter will usually involve the anterior cruciate ligament (ACL), the medical collateral ligament (MCL) and also the medial meniscus (MM) (cartilage on the inner aspect of the knee).
Ligament and Meniscus Injuries
Ligament injuries can be sustained either from external or internal forces. External forces will be your opponent (or an over zealous sparring partner!) and may come from a strike (to or below the joint) or a submission hold such as knee bar. Internal forces will be physiological stress from incorrect biomechanics such as not allowing the supporting leg to rotate during a round kick or during a clinch or a takedown. Any of these events may rupture not only the ACL, but may also damage the MCL and MM, totally disrupting the stability of the knee.
The ACL is injured during excessive knee rotation (hooking punches and round kicks) on the weight bearing knee if poor biomechanics come into play or during hyperextension (knee bar or locking out the knee on a badly executed kick). The MCL is injured by excessive force bending the knee inward (heavy kick or stamp to the outside of the knee or poor technique/biomechanics in sweeps and throws). The ACL, MCL and MM may all be injured together causing what is known as an “Unhappy triad”. Damage to the MM usually causes pain, swelling, joint locking, or the inability to completely straighten the joint due to a piece of meniscus physically blocking movement in the knee.
So how do you know if you’ve torn your ACL? Well if you feel a sharp pain in the knee, giving way and hear a sudden “pop” followed by immediate swelling with difficulty in full weight bearing then chances are you’ve torn your ACL! If however you only feel a tearing sensation chances are you’ve torn your MCL, not as bad as an ACL rupture….but still bad!
If you think you have suffered an injury to ligament or meniscus go to A&E; straight away and get an assessment of the damage. If you have torn your ACL surgical repair will probably be required followed by between 6 to 9 months of rehab before return to competition. Rehab is detailed and lengthy so a Chartered Physiotherapist will take you through your recovery from day 1 to return to full competition. MCL tears however, are often braced and left to heal without surgery whilst a tear of the meniscus may require arthroscopy (key-hole surgery).
Conservative (Non-surgical) Treatment of Knee Injuries
As always for acute knee injuries PRICE(MM) is standard practice. Protection, Rest, Ice, Compression and Elevation should be applied by the individual, then Medications and Modalities (physical treatments) should be sought from your GP or Chartered Physiotherapist. Bracing may also be used in the early stages. Rehabilitation (Surgical and conservative)
Some points that are important to consider during your rehab are:
Stretching: To maintaining joint range of motion as following injury or surgery as scar tissue forms and soft-tissues contract.
Strengthening: Exercises performed to rehabilitate the injured area back to competitive performance levels.
Proprioception: The ability to know where a body part is in space without looking at it. When proprioception is impaired following a ligament injury the joint may feel unstable which may increase the risk of re-injury. Proprioception training re-educates your body to control the position of an injured joint. It is best performed standing on the injured leg with the eyes closed and measure the amount of time you can remain stable. As you improve make it more difficult by standing on an unstable surface (eg. wobble board or trampette).
Better proprioception will improve your kicks, throws, takedowns and counters and overall balance.
Tip: Even if you are not injured train this aspect of your game; I’ve never worked with an athlete whose proprioception was too good!
Alternative training -Train Around the Injury
One of the major goals of rehabilitation is to maintain a your cardiovascular fitness levels, so for instance when you suffer a lower limb injury instead of roadwork, try aqua jogging with a flotation belt (running upright in a swimming pool without your feet touching the bottom of the pool). You, your coach/trainer and your physio should work together at devising alternative training programmes as soon as possible particularly if you are a competitive fighter. In addition to cardiovascular fitness, you may use the injury period as an opportunity to strengthen weaker areas whether they be physical, mental, technical or tactical.
Return to Training/Competition after Injury
Depending on the nature and severity of the injury, it may take many months of physical therapy for you return to full training or competition. The wide variety of knee injuries creates a wide range of recovery and rehab times with most ligament and meniscus injuries often taking months to rehabilitate whether surgery has taken place or not.
An ACL injury will keep you out of competition for 6 to 9 months. As with any injury, for return to full training/competition you must consider the risk of re-injury and the ability to fight/perform at a satisfactory level. To return to competition there must be a restoration of full strength, endurance, flexibility and proprioception. With repetitive injuries it is important to identify the specific activity that caused the initial injury so that activity can be avoided or training modified.
This is just a brief outline of the knee injuries you may incur during MMA training and competition and a rough guide to treatment and rehabilitation principles. If you have any specific knee problems you will need to seek first hand advice and treatment from an experienced sports injuries Chartered Physiotherapist.
This article is for the purpose of information only and it is not intended to diagnose or treat medical conditions and is not considered to be a substitute for individual medical assessment and advice.